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DOES THE "GAY GENE" REALLY EXIST? I have a friend who is a Christian. He was once a practicing homosexual. I asked him whether he believes in the "gay gene". He said he believes some people are born with a tendency to be gay, the way some people are born with a tendency to be alcoholics, but with God's help, they can overcome the tendency---the "gay gene". Do you believe in the "gay gene"? 1. I believe some people are born with a "gay gene", but God can help them be what He wants them to be. 2. I don't believe in the "gay gene". 3. I believe some people are born gay, and that's fine. 4. Other 
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Generational sins are cause by the curse of the law. The curse came in the earth with the fall of Adam. Exodus 20:5 Thou shalt not bow down thyself to them, nor serve them: for I the LORD thy God am a jealous God, visiting the iniquity of the fathers upon the children unto the third and fourth generation of them that hate me. Isaiah 42:8 " I am the Lord; that is my name! And My glory I will not give to another, nor My praise to graven images."
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Sin is the cause of abnormalities. Sin can be generational. So, some people are predisposed to certain sins heritically through the biological genetics because of the sins of the fathers. All mankind by nature are the children of wrath, born into sin. It is only by the miracle of the new birth that the body can be healed. For some gays whom God saves, this is instantaneous but for others it is a progressive work of the holy Spirit. One who is born gay cannot change that behavior without being redeemed. Much of Evangelical Christianity that adheres to arminian doctrine are constantly trying to impose the gospel on a sin sick world. Rather they should spend their time preaching the gospel and praying for God to save souls. Isaiah 42:8 " I am the Lord; that is my name! And My glory I will not give to another, nor My praise to graven images."
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This study only proves what the bible says. Man is born a sinner. A man does not sin and become a sinner but a sinner sins because HE IS A SINNER! This does not mean that a gay person cannot get saved but what it does mean is that it is impossible to save ones self. The sinner will never choose Jesus Christ as it is against their nature. Only the miracle of the new birth can save sinners from their sin. Ephesians 2:1-10 And you hath he quickened, who were dead in trespasses and sins; Wherein in time past ye walked according to the course of this world, according to the prince of the power of the air, the spirit that now worketh in the children of disobedience: Among whom also we all had our conversation in times past in the lusts of our flesh, fulfilling the desires of the flesh and of the mind; and were by nature the children of wrath, even as others. But God, who is rich in mercy, for his great love wherewith he loved us, Even when we were dead in sins, hath quickened us together with Christ, (by grace ye are saved  And hath raised us up together, and made us sit together in heavenly places in Christ Jesus: That in the ages to come he might shew the exceeding riches of his grace in his kindness toward us through Christ Jesus. For by grace are ye saved through faith; and that not of yourselves: it is the gift of God: Not of works, lest any man should boast. For we are his workmanship, created in Christ Jesus unto good works, which God hath before ordained that we should walk in them Isaiah 42:8 " I am the Lord; that is my name! And My glory I will not give to another, nor My praise to graven images."
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According to the American Psychological Association Most scientists today agree that sexual orientation is most likely the result of a complex interaction of environmental, cognitive and biological factors. In most people, sexual orientation is shaped at an early age. There is also considerable recent evidence to suggest that biology, including genetic or inborn hormonal factors, play a significant role in a person's sexuality. A number of biological factors have been considered by scientists, such as prenatal hormones, chromosomes, polygenetic effects, brain structure effects and even viral influences. Studies of how developmental mechanisms produce variations in sexual orientation ought to lead to a greater understanding of how many similar behavioral traits might work and develop. Empirical studies Twin studies Researchers have traditionally used twin studies to try to isolate genetic influences from environmental or other influences. Many early twin studies in this area selected from non-representative samples, and gave non-representative results.[4] Later twin studies have drawn from broader, more representative samples, and have thus given more representative results. A recent large-scale twin study, done by researchers at Yale University and Columbia University, concludes that "there is no evidence for strong genetic influence on same-sex preference in this sample." Earlier twin studies gave indications that male homosexuality was genetically mediated. One common type of twin study compared the monozygotic (or identical) twins of people possessing a particular trait to the dizygotic (non-identical, or fraternal) twins of people possessing the trait. Since monozygotic twins have the same genotype (genetic makeup) while dizygotic twins share only, on average, 50% of their genotype, a difference in the prevalence of the trait in question between these types of twins provides evidence of a genetic component. A few such studies began to examine homosexuality in the early 20th century, using small, non-random samples. Bailey and Pillard (1991) in a study of gay twins found that 52% of monozygotic brothers and 22% of the dizygotic twins were concordant for homosexuality.[6] Bailey, Dunne and Martin (2000) used the Australian twin registry to obtain a sample of 4,901 twins.[7] Self reported zygosity, sexual attraction, fantasy and behaviours were assessed by questionnaire and zygosity was serologically checked when in doubt. MZ twin concordance for homosexuality was found to be 30%. Criticism of these earlier studies included: recruitment through gay media (which may result in higher response rates from twins who are both gay), and recruitment from twin registries (which may result in higher response rates from twins who act more similar to each other[4]). Bearman & Bruckner (2002), by contrast, based their conclusions on a study drawn from a wide population.[4] The assessment of these researchers is: “ Among [identical] twins, 6.7% are concordant [that is, both express same-sex romantic attraction]. [Fraternal] twin pairs are 7.2% concordant. Full-siblings are 5.5% concordant. Clearly, the observed concordance rates do not correspond to degrees of genetic similarity. None of the comparisons between [identical] twins and others ... are even remotely significant. If same-sex romantic attraction has a genetic component, it is massively overwhelmed by other factors.[4] ” Estimates of heritability of homosexuality Study Male Female Hershberger, 1997 0% 48% Bailey et al., 2000 30% Kendler et al., 2000 28‒65% Kirk et al., 2000 30% 50‒60% Bearman et al., 2002 7.7% 5.3% Twin studies have received a number of criticisms including ascertainment bias where homosexuals with gay siblings are more likely to volunteer for studies. Another issue is the recent finding that even monozygotic twins can be different and there is a mechanism which might account for monozygotic twins being discordant for homosexuality. Gringas and Chen (2001) describe a number of mechanisms which can lead to differences between monozygotic twins, the most relevant here being chorionicity and amniocity. Dichorionic twins potentially have different hormonal environments and receive maternal blood from separate placenta. Monoamniotic twins share a hormonal environment, but can suffer from the 'twin to twin transfusion syndrome' in which one twin is "relatively stuffed with blood and the other exsanguinated".[9] If one twin receives less testosterone and the other more, this could result in different levels of brain masculinisation. Overall, data appear to indicate that genetic factors may play some part in the development of sexual orientation, even if only a modest part. Further work is needed to more precisely quantify any genetic contribution to sexuality and to elucidate its mechanism. Studies of brain structure A number of sections of the brain have been reported to be sexually dimorphic; that is, they vary between men and women. There have also been reports of variations in brain structure corresponding to sexual orientation. In 1990, Swaab and Hofman reported a difference in the size of the suprachiasmatic nucleus between homosexual and heterosexual men. In 1992, Allen and Gorski reported a difference related to sexual orientation in the size of the anterior commissure Early work of this type was also done by Simon LeVay - however, LeVay's assessments have not been replicated. LeVay studied four groups of neurons in the hypothalamus, called INAH1, INAH2, INAH3 and INAH4. This was a relevant area of the brain to study, because of evidence that this part of the brain played a role in the regulation of sexual behaviour in animals, and because INAH2 and INAH3 had previously been reported to differ in size between men and women.[12] He obtained brains from 41 deceased hospital patients. The subjects were classified as follows: 19 gay men who had died of AIDS, 16 presumed heterosexual men (6 of whom had died of AIDS), and 6 presumed heterosexual women (1 of whom had died of AIDS). The AIDS patients in the heterosexual groups were all identified from medical records as intravenous drug abusers or recipients of blood transfusions, though only 2 of the men in this category had specifically denied homosexual activity. The records of the remaining heterosexual subjects contained no information about their sexual orientation; they were assumed to have been mostly or all heterosexual "on the basis of the numerical preponderance of heterosexual men in the population." LeVay found no evidence for a difference between the groups in the size of INAH1, INAH2 or INAH4. However, the INAH3 group appeared to be twice as big in the heterosexual male group as in the gay male group; the difference was highly significant, and remained significant when only the 6 AIDS patients were included in the heterosexual group. The size of the INAH3 in the homosexual male brains was similar to that in the heterosexual female brains. However, he also found some contrary results: Three of the 19 homosexual subjects had a larger group of neurons in the hypothalamus than the average control-group subject. Three of the 16 control-group subjects had a smaller group of neurons in the hypothalamus than the average homosexual subject.[12] William Byne and colleagues attempted to replicate the differences reported in INAH 1-4 size using a different sample of brains from 14 HIV-positive homosexual males, 34 presumed heterosexual males (10 HIV-positive), and 34 presumed heterosexual females (9 HIV-positive). They found a significant difference in INAH3 size between heterosexual men and women. The INAH3 size of the homosexual men was apparently smaller than that of the heterosexual men and larger than that of the heterosexual women, though neither difference quite reached statistical significance. Byne and colleagues also weighed and counted numbers of neurons in INAH3, tests not carried out by LeVay. The results for INAH3 weight were similar to those for INAH3 size; that is, the INAH3 weight for the heterosexual male brains was significantly larger than for the heterosexual female brains, while the results for the gay male group were between those of the other two groups but not quite significantly different from either. The neuron count also found a male-female difference in INAH3, but found no trend related to sexual orientation. LeVay concluded in his 1991 paper that "The discovery that the nucleus differs in size between heterosexual and homosexual men illustrates that sexual orientation in humans is amenable to study at the biological level, and this discovery opens the door to studies of neurotransmitters or receptors that might be involved in regulating this aspect of personality. Further interpretation of the results of this study must be considered speculative. In particular, the results do not allow one to decide if the size of INAH 3 in an individual is the cause or consequence of that individual's sexual orientation, or if the size of INAH 3 and sexual orientation covary under the influence of some third, unidentified variable."He later added, "It's important to stress what I didn't find. I did not prove that homosexuality is genetic, or find a genetic cause for being gay. I didn't show that gay men are born that way, the most common mistake people make in interpreting my work. Nor did I locate a gay center in the brain. ... Since I look at adult brains, we don't know if the differences I found were there at birth or if they appeared later."[14] In addition to this, of the men LeVay used in his studies, the sexual histories of the "heterosexual" men were unknown. It is not clear from the research how HIV/AIDS may affect brain structure, a possible confounding factor. So rather than showing that differences in the neuron indicate homosexuality, LeVay's study may be showing that HIV/AIDS causes differences in neurons. It should be noted, however, that neither LeVay nor Byne found an HIV-related difference in INAH3 size. Chromosome linkage studies In 1993, Dean Hamer published findings from a linkage analysis of a sample of 76 gay brothers and their families. Hamer et al. (1993) found that the gay men had more gay male uncles and cousins on the maternal side of the family than on the paternal side.[15] Gay brothers who showed this maternal pedigree were then tested for X chromosome linkage, using twenty-two markers on the X chromosome to test for similar alleles. Thirty-three of the forty sibling pairs tested were found to have similar alleles in the distal region of Xq28, which was significantly higher than the expected rates of 50% for fraternal brothers. A later analysis by Hu et al. revealed that 67% of gay brothers in a new saturated sample shared a marker on the X chromosome at Xq28. Sanders et al. (199  replicated the study, finding 66% Xq28 marker sharing in 54 pairs of gay brothers.[17] These studies only examined homosexuality in males.However, two later studies (Bailey et al., 1999; McKnight and Malcolm, 2000) failed to find a preponderance of gay relatives in the maternal line of homosexual men. A study by Rice et al. in 1999 failed to replicate the Xq28 linkage.[18] More recently, Mustanski (2005) failed to find the Xq28 marker in a complete genome scan of gay men’s DNA. Mustanski did however find autosomal markers at 7q36, 8p12 and 10q26. The evidence for the Xq28 marker is therefore preliminary and has yet to be fully proved or disproved. Even at face value, the discovery of the Xq28 region would only show one genetic correlate of male homosexuality. Hamer's study was important though, as it was the first experiment to claim such a correlation. These findings do not suggest that the Xq28 region is necessary for homosexuality or singularly causes homosexuality, but rather that it might be one of many factors which influence sexual orientation in some males. A recent study supports X-linkage from a different perspective. Women have two X chromosomes, one of which is "switched off". The inactivation of the X chromosome occurs randomly throughout the embryo, resulting in cells that are mosaic with respect to which chromosome is active. In some cases though, it appears that this switching off can occur in a non-random fashion. Bocklandt et al (2006) reported that the number of women with extreme skewing of X chromosome inactivation is significantly higher in mothers of homosexual men than in age-matched controls without gay sons. 4% of controls showed extreme skewing compared to 13% of the mothers with gays sons and 23% of mothers with two or more gay sons.[20] Also, male homosexuality appears likely to be influenced by a complex genetic interaction which may be mediated by H-Y antigens in the mother’s immune system. Whichever genes are implicated they almost certainly cause male brains to differentiate in a female typical direction. As for female homosexuality, there remains little evidence from replicated genetic linkage studies. Maternal linkage, birth order, and female fertility Blanchard and Klassen (1997) reported that each older brother increases the odds of being gay by 33%.[21][22] This is now "one of the most reliable epidemiological variables ever identified in the study of sexual orientation." To explain this finding, it has been proposed that male fetuses provoke a maternal immune reaction that becomes stronger with each successive male fetus. Male fetuses produce HY antigens which are "almost certainly involved in the sexual differentiation of vertebrates." It is this antigen which maternal H-Y antibodies are proposed to both react to and 'remember'. Successive male fetuses are then attacked by H-Y antibodies which somehow decrease the ability of H-Y antigens to perform their usual function in brain masculinisation Bocklandt, Horvath, Vilain and Hamer (2006) reported that some mothers of gay babies have extreme skewing of X chromosome inactivation. Using a sample of 97 mothers of homosexual men and 103 mothers of heterosexual men, the pattern of X inactivation was ascertained from blood assays. 4% of the mothers of straight men showed extreme skewing compared to 13% of the mothers of gay men. Mothers of two or more gay babies had extreme skewing of X inactivation of 23%. This extreme skewing may influence male sexual orientation through the fraternal birth order effect. An alternate theory was proposed by Italian researchers in 2004 supported by a study of about 4,600 people who were the relatives of 98 homosexual and 100 heterosexual men. Female relatives of the homosexual men tended to have more offspring than those of the heterosexual men. Female relatives of the homosexual men on their mother's side tended to have more offspring than those on the father's side. The researchers concluded that there was genetic material being passed down on the X chromosome which both promotes fertility in the mother and homosexuality in her male offspring. The connections discovered, however, would explain only 20% of the cases studied, indicating that this might not be the sole genetic factor determining sexual orientation.[24] Homosexuals of either sex are more likely than the general population to be non-right handed. Pheromones correlation Recent research conducted in Sweden has suggested that gay and straight men respond differently to two odors that are believed to be involved in sexual arousal. The research showed that when both heterosexual women (lesbians were included in the study, but the results regarding them were "somewhat confused") and gay men are exposed to a testosterone derivative found in men's sweat, a region in the hypothalamus is activated. Heterosexual men, on the other hand, have a similar response to an estrogen-like compound found in women's urine. The conclusion, that sexual attraction, whether same-sex or opposite-sex oriented, operates similarly on a biological level, does not mean that there is necessarily a biological cause for homosexuality. Researchers have suggested that this possibility could be further explored by studying young subjects to see if similar responses in the hypothalamus are found and then correlating this data with adult sexual orientation Early fixation hypothesis Main article: Prenatal hormones and sexual orientation The early fixation hypothesis includes research into prenatal development and the environmental factors that control masculinization of the brain. Studies have concluded that there is empirical evidence to support this hypothesis, including the observed differences in brain structure and cognitive processing between homosexual and heterosexual men. One explanation for these differences is the idea that differential exposure to hormone levels in the womb during fetal development may block or exaggerate masculinization of the brain in homosexual men. The concentrations of these chemicals is thought to be influenced by fetal and maternal immune systems, maternal consumption of certain drugs, maternal stress, and direct injection. This hypothesis is also connected to the fraternal birth order research. Imprinting/critical period This type of theory holds that the formation of gender identity occurs in the first few years of life after birth. It argues that individuals can be predisposed to homosexual orientation by biological factors but are triggered in some cases by upbringing. Part of adopting a gender identity involves establishing the gender(s) of sexual attraction. This process is analogous to the "imprinting" process observed in animals. A baby duckling may be genetically programmed to "imprint" on a mother, but what entity it actually imprints upon depends on what objects it sees immediately after hatching. Most importantly, once this process has occurred, it cannot be reversed, any more than the duckling can hatch twice. A sort of reverse sexual imprinting has been observed in heterosexual humans. Several different triggers for imprinting upon a particular sexual orientation have been proposed. A common hypothesis, especially among non-scientists, is that something about what young children see in the gender-roles behavior of adults, or some differences (possibly unconscious) in the way adults treat young children, somehow influence or determine a child's eventual sexual orientation. This hypothesis, however, has not been supported by research findings that children of homosexuals are just as likely to be heterosexual as the general population and in reverse for children of heterosexuals in prevalence of homosexuality. Exotic becomes erotic Daryl Bem, a social psychologist at Cornell University, has theorized that the influence of biological factors on sexual orientation may be mediated by experiences in childhood. A child's temperament predisposes the child to prefer certain activities over others. Because of their temperament, which is influenced by biological variables such as genetic factors, some children will be attracted to activities that are commonly enjoyed by other children of the same gender. Others will prefer activities that are typical of another gender. This will make a gender-conforming child feel different from opposite-gender children, while gender-nonconforming children will feel different from children of their own gender. According to Bem, this feeling of difference will evoke physiological arousal when the child is near members of the gender which it considers as being 'different'. Bem theorizes that this physiological arousal will later be transformed into sexual arousal: children will become sexually attracted to the gender which they see as different ("exotic"). This theory is known as Exotic Becomes Erotic (EBE) theory. The theory is based in part on the frequent finding that a majority of gay men and lesbians report being gender-nonconforming during their childhood years. A meta-analysis of 48 studies showed childhood gender nonconformity to be the strongest predictor of a homosexual orientation for both men and women.[28] Fourteen studies published since Bailey & Zucker's 1995 also show the same results.[29] In one study by the Kinsey Institute of approximately 1000 gay men and lesbians (and a control group of 500 heterosexual men and women), 63% of both gay men and lesbians reported that they were gender nonconforming in childhood (i.e., did not like activities typical of their sex), compared with only 10-15% of heterosexual men and women. There are also six "prospective" studies--that is longitudinal studies that begin with gender-nonconforming boys at about age 7 and follow them up into adolescence and adulthood. These also show that a majority (63  of the gender nonconforming boys become gay or bisexual as adults.[30] There are very few prospective studies of gender nonconforming girls.[31][32] In a group of eighteen behaviorally masculine girls (mean age of assessment: 9 years), all reported a homosexual sexual orientation at adolescence, and eight had requested sex reassignment.[33]Politics The issue of genetic or other physiological determinants as the basis of sexual orientation is a highly politicised issue. The Advocate, a U.S. gay and lesbian newsmagazine, reported in 1996 that 61% of its readers believed that "it would mostly help gay and lesbian rights if homosexuality were found to be biologically determined". A cross-national study in the United States, the Philippines, and Sweden found that those who believed that "homosexuals are born that way" held significantly more positive attitudes toward homosexuality than those who believed that "homosexuals choose to be that way" and/or "learn to be that way". From Wikipedia, the free encyclopedia (for futher reading and more sources) [SIZE 5]Psalm 51:5 Behold, I was shapen in iniquity; and in sin did my mother conceive me Isaiah 42:8 " I am the Lord; that is my name! And My glory I will not give to another, nor My praise to graven images."
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It's nice to hear from someone who can feel for the people. Too often, we simply run over people. Thanks.
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I would like to have homosexuality be a non-issue. It would make life easier in some respects, but according to the Bible--Rom.1 is one place---it is a sin. So I must consider it a sin.
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4/7/2008 10:21 am |
You tell em, Sweetie. 
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Arminains would disagree because it disproves their doctrine that there is good in man and they can of their own free will choose God. Man BY NATURE IS A SINNER. BORN INTO SIN, SHAPED IN INIQUITYY, THE CHILDREN OF WRATH, THE CHILDREN OF THEIR FATHER THE DEVIL. Sniffing Out the Gay Gene By STEVEN PINKER Published: May 17, 2005 Cambridge, Mass. IT sounds like something out of the satirical journal Annals of Improbable Research: a team of Swedish neuroscientists scanned people's brains as they smelled a testosterone derivative found in men's sweat and an estrogen-like compound found in women's urine. In heterosexual men, a part of the hypothalamus (the seat of physical drives) responded to the female compound but not the male one; in heterosexual women and homosexual men, it was the other way around. But the discovery is more than just a shoo-in for that journal's annual Ig Nobel Prize - it raises provocative questions about the science and ethics of human sexuality. Scientists and perfume marketers who believe that humans, like other mammals, respond sexually to chemical signals called pheromones were cheered by the news. But we are a long way from dogs in heat. The role of pheromones in our sexuality must be small at best. When people want to be titillated or to check out a prospective partner, most seek words or pictures, not dirty laundry. The difference in the brain responses of gay and straight men does not, by itself, prove that homosexuality is innate; after all, learned inclinations, like innate ones, must reside somewhere in the brain. But in this case nature probably does trump nurture. Gay men generally report that their homosexual attractions began as soon as they felt sexual stirrings before adolescence. And homosexuality is more concordant in identical than in fraternal twins, suggesting that their shared genes play a role. Homosexuality is a puzzle for biology, not because homosexuality itself is evolutionarily maladaptive (though no more so than any other sexual act that does not result in conception), but because any genetic tendency to avoid heterosexual opportunities should have been selected out long ago. Perhaps "gay genes" have some other compensating advantage, like enhancing fertility, when they are carried by women. Perhaps the environments that set off homosexuality today didn't exist while our genes were being selected. Or perhaps the main cause is biological yet not directly genetic, like differences in hormones or antibodies that affect the fetus while it is developing. Just as puzzling is the existence of homophobia. Why didn't evolution shape straight men to react to their gay fellows by thinking: "Great! More women for me!" Probably the answer lies in a cross-wiring between our senses of morality and disgust. People often confuse their own revulsion with objective sinfulness, as when they dehumanize people living in squalor or, in the other direction, engage in religious rituals of cleanliness and purification. An impulse to avoid homosexual contact may blur into an impulse to condemn homosexuality. Cultural conservatives like the talk-show host Dr. Laura Schlesinger ostensibly condemn homosexuality for another reason - that it is a "biological error." Actually, it is she who has made the biological error. What is evolutionarily adaptive and what is morally justifiable have little to do with each other. Many laudable activities - being faithful to one's spouse, turning the other cheek, treating every child as precious, loving thy neighbor as thyself - are "biological errors" and are rare or unknown in the natural world. It's not just anti-gay commentators who see a moral coloring in the biology of homosexuality. Some gay groups condemn such research because it could stigmatize gay people as defective and lead to a day in which parents could selectively abort children with "gay genes." Others welcome the research because it shows that people don't "choose" to be gay and hence can't be criticized for it, nor could homosexuals convert the children in their classrooms or Scout troops even if they wanted to. It may not be a coincidence that the new discovery came from researchers in Europe. In America, the biology of homosexuality is a politicized minefield that scares away scientists (and the universities and agencies that pay for their research). Which is a pity. Regardless of where homosexuality resides in the brain, the ethics of homosexuality is a no-brainer: what consenting adults do in private is nobody's business but their own. And the deterrents to research on homosexuality leave us in ignorance of one of the most fascinating sources of human diversity. Steven Pinker, a professor of cognitive science at Harvard, is the author of "How the Mind Works" and "The Blank Slate." NEW YORK TIMES King James Version: Romans Chapter 1 1 Paul, a servant of Jesus Christ, called to be an apostle, separated unto the gospel of God, 2 (Which he had promised afore by his prophets in the holy scriptures,) 3 Concerning his Son Jesus Christ our Lord, which was made of the seed of David according to the flesh; 4 And declared to be the Son of God with power, according to the spirit of holiness, by the resurrection from the dead: 5 By whom we have received grace and apostleship, for obedience to the faith among all nations, for his name: 6 Among whom are ye also the called of Jesus Christ: 7 To all that be in Rome, beloved of God, called to be saints: Grace to you and peace from God our Father, and the Lord Jesus Christ. 8 First, I thank my God through Jesus Christ for you all, that your faith is spoken of throughout the whole world. 9 For God is my witness, whom I serve with my spirit in the gospel of his Son, that without ceasing I make mention of you always in my prayers; 10 Making request, if by any means now at length I might have a prosperous journey by the will of God to come unto you. 11 For I long to see you, that I may impart unto you some spiritual gift, to the end ye may be established; 12 That is, that I may be comforted together with you by the mutual faith both of you and me. 13 Now I would not have you ignorant, brethren, that oftentimes I purposed to come unto you, (but was let hitherto,) that I might have some fruit among you also, even as among other Gentiles. 14 I am debtor both to the Greeks, and to the Barbarians; both to the wise, and to the unwise. 15 So, as much as in me is, I am ready to preach the gospel to you that are at Rome also. 16 For I am not ashamed of the gospel of Christ: for it is the power of God unto salvation to every one that believeth; to the Jew first, and also to the Greek. 17 For therein is the righteousness of God revealed from faith to faith: as it is written, The just shall live by faith. 18 For the wrath of God is revealed from heaven against all ungodliness and unrighteousness of men, who hold the truth in unrighteousness; 19 Because that which may be known of God is manifest in them; for God hath shewed it unto them. 20 For the invisible things of him from the creation of the world are clearly seen, being understood by the things that are made, even his eternal power and Godhead; so that they are without excuse: 21 Because that, when they knew God, they glorified him not as God, neither were thankful; but became vain in their imaginations, and their foolish heart was darkened. 22 Professing themselves to be wise, they became fools, 23 And changed the glory of the uncorruptible God into an image made like to corruptible man, and to birds, and fourfooted beasts, and creeping things. 24 Wherefore God also gave them up to uncleanness through the lusts of their own hearts, to dishonour their own bodies between themselves: 25 Who changed the truth of God into a lie, and worshipped and served the creature more than the Creator, who is blessed for ever. Amen. 26 For this cause God gave them up unto vile affections: for even their women did change the natural use into that which is against nature: 27 And likewise also the men, leaving the natural use of the woman, burned in their lust one toward another; men with men working that which is unseemly, and receiving in themselves that recompence of their error which was meet. 28 And even as they did not like to retain God in their knowledge, God gave them over to a reprobate mind, to do those things which are not convenient; 29 Being filled with all unrighteousness, fornication, wickedness, covetousness, maliciousness; full of envy, murder, debate, deceit, malignity; whisperers, 30 Backbiters, haters of God, despiteful, proud, boasters, inventors of evil things, disobedient to parents, 31 Without understanding, covenantbreakers, without natural affection, implacable, unmerciful: 32 Who knowing the judgment of God, that they which commit such things are worthy of death, not only do the same, but have pleasure in them that do them. Isaiah 42:8 " I am the Lord; that is my name! And My glory I will not give to another, nor My praise to graven images."
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Scientifically there is zero evidence that a gay gene exists period Being queer is a choice. It is called an abomination in the scriptures, not just sin. God would not make anyone like this. I have many friends over the years that are effeminate and have many children they have fathered. In the word it speaks of giving ourselves over to a reprobate mind. It is a perversion of the natural.In the mind and physical So your friend, over time gave themselves over to a reprobate mind. There have been many lies over the years such as brains being formed dfferently. It is a bad spiritual condition. Lets look at pole dancers, or strippers if you please. Were women born with a desire to take their clothes off in front of strange men? Of course not. But over a period of time they do things and become desensitized to the point it bothers them very little. It is a similar process with queer situations I do not like to use the word gay. When I grew up it meant something nice. Perhaps I should use the word sodomite. It better describes it.There is nothing beautiful about it.
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4/7/2008 12:04 am |
I must say Mia is correct as i don't believe that either, it is the society we live in that accepts all that lifestyle stuff. Gods way is my way,...Clay
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lol...I know several guys named Gene, and every time I typed "gay gene" I wondered what they would say. Great minds think alike.
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